Welders Pneumoconiosis

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What is Welders Pneumoconiosis

Welders pneumoconiosis (welder's pneumoconiosis) is a pneumoconiosis mainly chronic fibrous hyperplasia of the lung tissue damage caused by long-term inhalation of high concentrations of welding fumes.

Symptoms of Welders Pneumoconiosis

Welders pneumoconiosis slow, the onset worked for more than 10 years, range 15 to 20 years. The shortest onset seniority for four years. Welders pneumoconiosis clinical symptoms are mild: there are clear signs of X-ray may be no obvious symptoms and signs. With the progress of the disease, especially emphysema, bronchiectasis, or bronchitis, there may be clinical symptoms. Welders early, lung function is often the case when the normal range, emphysema and other diseases, lung function was reduced accordingly. The general course of the slow progress, a small number of cases from the welding operation, the condition can gradually reduce. Has long been engaged in the job of exposure to high concentrations of welding smoke, a few cases may also occur Ⅲ of pneumoconiosis.

What Causes Welders Pneumoconiosis

The welding technology is indispensable in modern industrial process, welding process many types HSBC have manual metal arc welding, self-shielded welding, carbon dioxide arc welding, TIG, submerged arc welding and carbon arc gouging. The Welders pneumoconiosis called, is an arc welding electrode coating, the core wire and the metal being welded in the arc under high temperature melting, evaporation and oxidation to produce a large number of metal oxides and other soot oven in air.

Long-term exposure to high concentrations of welding fumes, welding work in airtight container or poorly ventilated environment, can cause welders pneumoconiosis.

Tests and Diagnosis for Welders Pneumoconiosis

The grayish black lungs, the lungs can be seen scattered in the different sizes of the dust stove welders pneumoconiosis identification, dust lesions with irregular-shaped or stellate-shaped, a small number were round. Dust stove diameter multi 1mm or less minority to 1 to 2 mm in diameter, up to 3mm in diameter rarely. Most dust foci collagen fiber content at 50% or less, and the portion of the dust stove for simple dust calm, excluding or containing a small amount of collagen fibers, in the form of a dust spot exists. The dust spots distribution in the alveolar space, alveolar asked traps, respiratory bronchioles and perivascular. Dust particles iron staining was positive, the slice high temperature ashing not found birefringent quartz dust particles. The intrapulmonary visible scattered distribution of about 2mm nodules, some nodules can be dense piles, quality and tough, nodules were seen large amounts of coarse collagen fibers, may also occur hyalinization. Welders pneumoconiosis diagnosis often can be seen in the dust spots and nodules around the different levels of the perifocal emphysema. Few welders pneumoconiosis autopsy proved the intrapulmonary visible chunks constituted by the the intensive large amounts of dust fibers stove and extensive interstitial fibrosis pulmonary fibrosis. Due to the role of welding fumes and oxides of nitrogen and other harmful gases, lung size bronchial dilation and inflammation. Diagnosis:

X-ray findings of early irregularly shaped small shadows was both lungs diffuse distribution, small circular shadow appeared later, p shadow, and widely distributed, low intensity, the intensity with the progression of the disease The gradual increase in the individual advanced cases, a large shadow. Signs of hilar generally do not increase, few pleural adhesions and emphysema, the small number of cases increased hilar density, increase the shadow structural disorder. Out of a job, there is little progress.

Treatments of Welders Pneumoconiosis

Welders pneumoconiosis therapy:

1 from the welding work arrangements for light work, regular follow-up review.

2 without antifibrotic therapy.

3 a symptomatic treatment.